Activate your 30 day free trialto continue reading. In the Tips for Living Well with COPD flyer, you will learn about tools, techniques, and resources to help you on your journey with COPD. COPD - Physical Findings. You can read the details below. Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. Number of Views: 680. Presentation Transcript. 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By accepting, you agree to the updated privacy policy. COPD COMPLETE POWER POINT AS PER GOLD. chronic obstructive pulmonary disease in equine, COPD (Chronic obstructive Pulmonary Disease) PowerPoint Presentation -aslam, Chronic obstructive pulmonary disease (copd) power point, Nursing care plans, concept map bronhial asthma, Introduction & investigations to respiratory diseases, L'Docile - Respiratory diseases & nebulization Report, J. Parker Emphysema Presentation Powerpoint, COPD - Chronic Obstructive Pulmonary Disease |medico X| Pathology. Description: COPD 'COPD is fifth commonest cause of death in England and Wales accounting for . Decreased quality of life. Quieres ms informacin?Llame al 866.731.COPD (2673) y presione 9 para hablar en espaol con un paciente o cuidador. Nursing Times [online]; 116: 4, 27-30. . Copd 1. Chronic obstructive pulmonary disease (COPD) is a long-term lung disease that involves the obstruction of airflow due to an inflammation of the lungs. COPD: CHRONIC OBSTRUCTIVE PULMONARY DISEASE Callie Cluf WHAT IS COPD: COPD is a common pulmonary disease that. MDA, Interventional Pulmonary Course 2023, 2/23/2023 7:00:00 AM - 2/24/2023 4:45:00 PM, The activity is designed to review the current concepts and recent advances in interventional pulmonology as it pertains to the cancer patient, and to introduce new technologic advances that are poised to revolutionize the diagnosis, staging, and management of lung cancer. It appears that you have an ad-blocker running. Weve updated our privacy policy so that we are compliant with changing global privacy regulations and to provide you with insight into the limited ways in which we use your data. . This plan guides you when youre feeling well and when an exacerbation (flare-up) is starting. We've updated our privacy policy. . INTRODUCTION And/or Chronic Obstructive Pulmonary Disease . Energy-conserving tips are presented. It can be very helpful to have an action plan to follow when managing your COPD. Epidemiology Age Higher prevalence with increasing age Doseresponse relationship between cigarette smoking intensity and decreased pulmonary function, Risk Factors Cigarette smoking is a major risk factor. review oct. 16, 2014 cathy vakil. She is a passionate nurse, educator and researcher, with research interests in undergraduate nursing education, nursing workforce, chronic illness experience, patient education, health . . Bandaged Teddy Bear - Nursing Templates Free. Slide 3-. The Guides are available for purchase individually or in a set in our online catalogue. However, people whose disease progresses to more severe stages . Many in this series of 12- to 20 . Enhanced Discharge Process to Reduce 30-Day COPD Readmission Rates: A Quality Improvement Project. Its important for individuals with COPD to stay healthy and enjoy their favorite activities. This one-page fact sheet focuses on preventing pneumonia in individuals with COPD. This page was reviewed on January 10, 2023 by the COPD Foundation Content Review and Evaluation Committee. This n urse PowerPoint template for free comes with many medical illustrations. 527 Views Download Presentation. Blood gases drawn on room air revealed these values: patient 1- pH =7.18, PCO2 = 70mmHg, PO2=50mmHg, HCO3=24mEq/L; patient2- pH =7.31, PCO2=50mmHg, PO2=50mmHg, HCO3=25mEq/L, Comment The A-a gradient calculation for patient 1 is as follows: A-a DO2 = PAO2 PaO2 PAO2 = 150 (1.25x PCO2) PAO2 = 150 (1.25x 70) PAO2 = 62 A-a =62 50 A-a = 12. Background: Qualitative research on long COVID by subtype has not yet occurred. Chronic obstructive pulmonary diseases & Nursing care. This flyer will give you helpful tips and tools for staying healthy, avoiding illness, and recognizing early warning signs of an infection or flare-up. Comment We can be reasonably confident that patient 1 suffered hypoventilation due to the effect of the ingested drugs on the brain stem. Log in. *ctda0&?iOcdr0&O@(L%EUxmsy]r@y9`P PPT layouts enhanced with lung - senior woman gets first aid background and a . Free access to premium services like Tuneln, Mubi and more. 28-7, Emphysema Pathophysiology
Hyperinflation of alveoli
Destruction of alveolar walls
Destruction of alveolar capillary walls
Narrowed airways
Loss of lung elasticity, Emphysema Pathophysiology Two types:
Centrilobular (central part of lobule)
Most common
Panlobular (destruction of whole lobule)
Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are:
Hyperinflation of alveoli
Destruction of alveolar capillary walls
Narrowed, tortuous small airways
Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of
Mucus
Smooth muscle spasm
Inflammatory process
Collapse of bronchiolar walls
Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction
inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed
Air goes into the lungs but is unable to come out on its own and remains in the lung
Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention
As more alveoli coalesce, blebs and bullae may develop
Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion
Compensation is done by increasing respiratory rate to increase alveolar ventilation
Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations
Dyspnea
Progresses in severity
Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum
Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations
Patient becomes chest breather, relying on accessory muscles
Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are:
Hyperplasia of mucus-secreting glands
in trachea and bronchi
Increase in goblet cells
Disappearance of cilia
Chronic inflammatory changes and narrrowing of small airways
Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation
Primary pathologic mechanism causing changes
Narrow airway lumen and reduced airflow d/t
hyperplasia of mucus glands
Inflammatory swelling
Excess, thick mucus, Chronic Bronchitis Pathophysiology
Greater resistance to airflow increases work of breathing
Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation
Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion
Tendency to hypoventilate and retain CO2
Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions
Bronchospasm frequently develops
More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms:
Frequent, productive cough during winter
Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing
Cough
Dyspnea on exertion
History of smoking
Normal weight or heavyset
Ruddy (bluish-red) appearance d/t
polycythemia (increased Hgb d/t chronic hypoxemia))
cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia
Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis)
Cor pulmonale (Rt heart hypertrophy + RV failure)
Pneumonia
Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities
History and physical exam
Pulmonary function studies
reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs
PaO2
PaCO2 (especially in chronic bronchitis)
pH (especially in chronic bronchitis)
Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation
Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy
Bronchodilators as maintenance therapy
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